Last edited by Mejin
Saturday, May 16, 2020 | History

2 edition of Apoptosis: Involvement of Oxidative Stress and Intracellular Ca2+ Homeostasi found in the catalog.

Apoptosis: Involvement of Oxidative Stress and Intracellular Ca2+ Homeostasi

by Gines Maria Salido

  • 142 Want to read
  • 28 Currently reading

Published by Springer Netherlands in Dordrecht .
Written in English

    Subjects:
  • Medicine,
  • Life sciences,
  • Cytology

  • Edition Notes

    Statementedited by Gines Maria Salido, Juan Antonio Rosado
    ContributionsRosado, Juan Antonio, SpringerLink (Online service)
    The Physical Object
    Format[electronic resource] /
    ID Numbers
    Open LibraryOL25565671M
    ISBN 109781402098727, 9781402098734

    Oxidative stress is one of the most potent activators of ASK1, which is essential for oxidative stress-induced apoptosis through the activation of MKK4/MKK7-JNK cascade Oxidative stress induces phosphorylation of Thr in the activation loop of ASK1, correlating with enhanced ASK1 activity and increased apoptosis 65 (Fig. 5).Cited by: Ionizing radiation (IR) causes an increase in intracellular calcium, alters contractility, and triggers apoptosis via the activation of PKCα and -ε in irradiated smooth muscle cells. The present study investigated the role of the mitochondria in these processes and characterized the proteins involved in IR-induced apoptosis. Intestinal smooth muscle cells were exposed to 10–50 Gy from a γ Author: Sandra Claro, Alice Teixeira Ferreira, Maria Etsuko Miyamoto Oshiro.

    Ca2+ homeostasis and the plasma membrane Na+/Ca2+ exchanger are vital to many cellular functions and physiological processes. This volume, which is the proceedings of the fifth in a series of international conferences, includes contributions that merge molecular biology, biochemistry, biophysics, and physiology, providing novel insights that significantly advance our understanding of Na+/Ca2.   Abstract. The dominant model for Alzheimer’s disease (AD) is the amyloid cascade hypothesis, in which the accumulation of excess amyloid-β (Aβ) leads to inflammation, excess glutamate and intracellular calcium, oxidative stress, tau hyperphosphorylation and tangle formation, neuronal loss, and ultimately by: 2.

    [] Samhan-Arias AK, Marques-da-Silva D, Yanamala N, Gutierrez-Merino C. Stimulation and clustering of cytochrome b(5) reductase in caveolin-rich lipid microdomains is an early event in oxidative stress-mediated apoptosis of cerebellar granule neurons. J Proteomics ; Autophagy and the ubiquitin–proteasome system (UPS) are the two major intracellular quality control and recycling mechanisms that are responsible for cellular homeostasis in eukaryotes. Ubiquitylation is utilized as a degradation signal by both systems, yet, different mechanisms are in play. The UPS is responsible for the degradation of short-lived proteins and soluble misfolded proteins Cited by:


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Apoptosis: Involvement of Oxidative Stress and Intracellular Ca2+ Homeostasi by Gines Maria Salido Download PDF EPUB FB2

Apoptosis: Involvement of Oxidative Stress and Intracellular Ca 2+ Homeostasis, presents a concise synthesis of the current knowledge and recent advances in the mechanisms of apoptosis in different cells and the role of oxidative stress and Ca 2+ signalling.

Particular attention is given to the different features of apoptosis in distinct cell types, ranging from hepatocytes to cardiovascular Format: Hardcover. Apoptosis: Involvement of Oxidative Stress and Intracellular Ca2+ Homeostasis, presents a concise synthesis of the current knowledge and recent advances in the mechanisms of apoptosis in different cells and the role of oxidative stress and Ca2+ signalling.

Particular attention is given to the. Apoptosis: Involvement of Oxidative Stress and Intracellular Ca 2+ Homeostasis, presents a concise synthesis of the current knowledge and recent advances in the mechanisms of apoptosis in different cells and the role of oxidative stress and Ca 2+ signalling.

Particular attention is given to the different features of apoptosis in distinct cell types, ranging from hepatocytes to cardiovascular. Get this from a library. Apoptosis: involvement of oxidative stress and intracellular Ca²+ homeostasis.

[Gines Maria Salido; Juan Antonio Rosado;] -- "Apoptosis: Involvement of Oxidative Stress and Intracellular Ca2+ Homeostasis, presents a concise synthesis of the current knowledge and recent advances in the mechanisms of apoptosis in different.

The book is divided in two major chapter sections: general mechanisms of the apoptotic pathways and the role of oxidative stress and intracellular Ca2+ homeostasis and a more specific section dedicated to the specificities of apoptosis in a number of excitable and non-excitable cells.

Get this from a library. Apoptosis: involvement of oxidative stress and intracellular Ca² homeostasis. [Gines Maria Salido; Juan Antonio Rosado;] -- Presents a synthesis of knowledge and advances in the mechanisms of apoptosis in different cells and the role of oxidative stress and Ca2+ signalling.

This book focuses on the different features of. from book Apoptosis: Involvement of Oxidative Stress and Intracellular Ca2+ Homeostasi (pp) Oxidative Stress, Intracellular Calcium Signals and Apoptotic Processes Chapter August with.

Apoptosis: Involvement of Oxidative Stress and Intracellular Ca2+ Homeostasis This book revisits the modeling of bioreactors using a multi-scale approach. It addresses issues related to mixing, phase-to-phase transfers and the adaptation of microorganisms to variations in concentration, and explores the use of population balances for the.

from book Apoptosis: Involvement of Oxidative Stress and Intracellular Ca2+ Homeostasi (pp) Apoptosis in the Liver Chapter January with 85 Reads. Salido G. () Oxidative Stress, Intracellular Calcium Signals and Apoptotic Processes.

In: Salido G.M., Rosado J.A. (eds) Apoptosis: Involvement of Oxidative Stress and Intracellular Ca2+ by: 9. The book presents a concise synthesis of the current knowledge and recent advances in the mechanisms of apoptosis in different cells and the role of oxidative stress and Ca2+ signalling.

Particular attention is given to the different features of apoptosis in distinct cell types, ranging from hepatocytes to cardiovascular and blood cells. All of these studies point to the importance of ROS as inducers of cell death and argue for the relevance of having mechanisms of protection against oxidative insults.

Ca 2§ Homeostasis Apoptosis is also induced by disturbing the homeostasis of intracellular Ca 2§ concentration ([Ca2+]i). Title:The Role of ER Stress-Induced Apoptosis in Neurodegeneration VOLUME: 9 ISSUE: 3 Author(s):Ioanna C.

Stefani, Daniel Wright, Karen M. Polizzi and Cleo Kontoravdi Affiliation:Department of Chemical Engineering, Imperial College London, London SW7 2AZ, U.K.

Keywords:Apoptosis, calcium dyshomeostasis, endoplasmic reticulum stress, neurodegeneration, oxidative stress, reactive oxygen. A number of disorders, such as Alzheimer disease and diabetes mellitus, have in common the alteration of the redox balance, resulting in an increase in reactive oxygen species (ROS) generation that might lead to the development of apoptosis and cell death.

It has long been known that ROS can significantly alter Ca2+ mobilization, an intracellular signal that is involved in the regulation of a Cited by: G.M. Salido, Oxidative Stress, Intracellular Calcium Signals and Apoptotic Processes, Apoptosis: Involvement of Oxidative Stress and Intracellular Ca2+ Homeostasi, / Cited by:   Accumulating data suggest that oxidative stress and mitochondrial damage are involved in the pathogenesis of neurodegenerative diseases, and antioxidant administration to modify mitochondrial dysfunction may be useful in the prevention and treatment of neurodegenerative diseases[,].

Mitochondria are a major source of intracellular Cited by: The book is splendidly illustrated with up-to-date radiographs, MDCT CT scans, and multiplanar CT, CTangiographic and some MR and 3-D imaging. More than 1, high-quality images make the reading easy and captions are concise and significant.

The text is outstanding and straightforward. -- Pediatric Radiology Written by renowned experts in chest. Bcl-2 proteins are major regulators of cellular responses to intrinsic and extrinsic apoptotic stimuli.

Among them, overexpression of the antiapoptotic protein Bcl-x L modulates intracellular Ca 2+ homeostasis and organelle-specific apoptotic signaling pathways. However, the specific activities of Bcl-x L at mitochondria and the endoplasmic reticulum (ER) have not been fully defined.

Death receptor 6 (DR6), also known as tumor necrosis factor receptor superfamily member 21 (TNFRSF21), is a cell surface receptor of the tumor necrosis factor receptor superfamily which activates the JNK and NF-κB pathways.

It is mostly expressed in the thymus, spleen and white blood cells. The Gene for DR6 is 78, bases long and is found on the 6th s: TNFRSF21, BM, CD, DR6, tumor. Disruption of intracellular Ca 2+ homeostasis, or induction of mild oxidative stress, might mediate the apoptosis-inducing effects of some of these chemicals.

Activation of cell death might also contribute to the toxicity of certain representatives of the rapidly growing class of Cited by:.

Oxidative stress is a well-established event in the pathology of several neurobiological diseases. Sirt3 is a nicotinamide adenine nucleotide (NAD+)-dependent protein deacetylase that regulates mitochondrial function and metabolism in response to caloric restriction and stress.

This study aims to investigate the role of Sirt3 in H2O2 induced oxidative neuronal injury in primary cultured rat Cited by:   Reactive Oxygen Species (ROS), Nanoparticles, and Endoplasmic Reticulum (ER) Stress-Induced Cell Death Mechanisms presents the role of ROS‒mediated pathways cellular signaling stress, endoplasmic reticulum (ER) stress, oxidative stress, oxidative damage, nanomaterials, and the mechanisms by which metalloids and nanoparticles induce their toxic Edition: 1.Y.

Gutierrez-Martin, F. J. Martin-Romero, F. Henao, and C. Gutierrez-Merino, “Alteration of cytosolic free calcium homeostasis by SIN high sensitivity of L-type Ca2+ channels to extracellular oxidative/nitrosative stress in cerebellar granule cells,” Journal Cited by: 3.